Fructose-Asparagine Is a Primary Nutrient during Growth of Salmonella in the Inflamed Intestine

نویسندگان

  • Mohamed M. Ali
  • David L. Newsom
  • Juan F. González
  • Anice Sabag-Daigle
  • Christopher Stahl
  • Brandi Steidley
  • Judith Dubena
  • Jessica L. Dyszel
  • Jenee N. Smith
  • Yakhya Dieye
  • Razvan Arsenescu
  • Prosper N. Boyaka
  • Steven Krakowka
  • Tony Romeo
  • Edward J. Behrman
  • Peter White
  • Brian M. M. Ahmer
چکیده

Salmonella enterica serovar Typhimurium (Salmonella) is one of the most significant food-borne pathogens affecting both humans and agriculture. We have determined that Salmonella encodes an uptake and utilization pathway specific for a novel nutrient, fructose-asparagine (F-Asn), which is essential for Salmonella fitness in the inflamed intestine (modeled using germ-free, streptomycin-treated, ex-germ-free with human microbiota, and IL10-/- mice). The locus encoding F-Asn utilization, fra, provides an advantage only if Salmonella can initiate inflammation and use tetrathionate as a terminal electron acceptor for anaerobic respiration (the fra phenotype is lost in Salmonella SPI1- SPI2- or ttrA mutants, respectively). The severe fitness defect of a Salmonella fra mutant suggests that F-Asn is the primary nutrient utilized by Salmonella in the inflamed intestine and that this system provides a valuable target for novel therapies.

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عنوان ژورنال:

دوره 10  شماره 

صفحات  -

تاریخ انتشار 2014